Yacob Mathai Kunnathazhath

Marma Health Centre,
India

Abstract Title:Baseless fever research

Biography:

A practicing physician in the field of healthcare in the state of Kerala in India for the last 36 years and very much interested in basic research. My interest is spread across the fever, inflammation and back pain. I am a writer. I already printed and published ten books on these subjects. I wrote hundreds of articles in various magazines. I have published 11 articles on fever in various journals. After scientific studies, we have developed 8000 affirmative cross checking questions. It can explain all queries related to fever.

Research Interest:

Baseless fever research. Lack of a uniform definition of fever and hyperthermia1 and lack of knowledge about the purpose of fever temperature lead to prescribing antipyretics for fever as for hyperthermia. It is often forgotten that increased inflammation increases the risk of infection and death2. So far, fever research has tested and treated fever by creating hyperthermia, the opposite of fever. Many research studies use hot objects or heating materials, such as electric bulbs and prostaglandinE2 to induce fever in laboratory animals such as rats and rabbits. Fever is determined by observing the rise in temperature in these animals using a thermometer. It is not a fever-measuring device. Little did they know that what these researchers were producing and testing was not fever, but hyperthermia? In many studies, decreased blood flow and abnormalities due to inflammation have been implicated as abnormalities due to increased fever. For example, Research shows that if a diabetic patient has a fever, both diabetes and inflammation increase. Fever causes fits, constricts blood vessels, lowers blood pressure, stimulates cold-sensing C neurons, and inhibits heat-sensing W (W) neurons. The fundamental fallacy of fever begins here and is reflected in the definition of fever and continues in diagnosis and treatment. Here, what happens when inflammation reduces blood flow in diabetic patients, do fits occur when inflammation decreases blood flow to the brain? Do blood vessels constrict when blood flow is reduced due to inflammation in the body? Does inflammation lower blood pressure? Does inflammation excite cold-sensing C neurons, and inhibit heat-sensing W (W) neurons? That’s all there is to research. No researcher who has studied fever has identified prostaglandin E2 deficiency as a cause of inflammation, infection, or death. Nowhere in fever research to date, nor current fever testing or treatment, has anyone addressed, examined, or treated why fever shows all the symptoms, signs, and actions of hypothermia caused by decreased temperature and decreased blood flow. Studies and research are based on the misconception that fever is the cause of all problems instead of disease. The causes, diagnosis and treatment of fever are fundamentally based on misinformation. Based on this, no matter how many billions of fever experiments have been done over the centuries, it is not about the right fever. Therefore, the correct result is not obtained. Similarly, fever and hyperthermia, being from opposite sources and opposite to each other, cannot be checked with the same substances and treated with the same substances. Keywords: uniform definition, prostaglandin E2, fever, immune system, inflammation, Fever research